Causes and Development of PTSD

Post-Traumatic Stress Disorder (PTSD) is a complex mental health condition that can develop after experiencing or witnessing traumatic events. While trauma is common, not everyone exposed to trauma develops PTSD, pointing to intricate biological, psychological, and social factors behind the disorder’s development.
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FREQUENTLY ASKED QUESTIONS

Yes. PTSD symptoms often appear within 3 months, but in some cases, they may emerge months or years after the traumatic event.
No. Most people exposed to trauma do not develop PTSD. Resilience factors often protect individuals from long-term consequences.
Yes. Those with prior trauma, mental health conditions, or lack of social support are at higher risk.
PTSD can cause persistent brain changes, but neuroplasticity means the brain can adapt and recover with treatment.

KEY TERMS

Brain Structure and Function

Neuroimaging studies reveal key brain differences in people with PTSD:

  • Amygdala: Overactive, leading to heightened fear responses.

  • Hippocampus: Reduced volume, impairing memory processing and context discrimination.

  • Prefrontal Cortex: Underactive, affecting emotional regulation and decision-making.

These changes contribute to persistent fear, flashbacks, and exaggerated responses to triggers.

Genetics and Heritability

Genetic factors contribute to PTSD vulnerability:

  • Twin studies show heritability estimates between 30-40% (NIMH, 2024).

  • Variations in genes regulating stress hormones, like FKBP5, have been linked to increased PTSD risk.

However, genetics alone don’t determine who develops PTSD. Environmental factors remain crucial.

Hormonal Responses

Trauma alters the body’s hypothalamic-pituitary-adrenal (HPA) axis, the stress hormone system:

  • People with PTSD often show abnormal cortisol levels.

  • Excessive norepinephrine leads to hypervigilance and sleep disturbances.

These biological changes help explain the persistent anxiety and physical symptoms seen in PTSD.

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